Role of oxidative stress in diabetic complications

Oxidative stress results from increased ROS and/or reactive nitrogen species (RNS). Examples of ROS include charged species such as superoxide and the hydroxyl radical, and uncharged species such as hydrogen peroxide and singlet oxygen. The possible sources of oxidative stress in diabetes might include auto-oxidation of glucose, shifts in redox balances, decreased tissue concentrations of low molecular weight antioxidants, such as reduced glutathione (GSH) and vitamin E, and impaired activities of antioxidant defenses enzymes such as superoxide dismutase (SOD) and catalase (CAT).  The oxidative stress plays a main factor in diabetic complications because the oxidative stress lead to the whole body damage and the vitamin deficiency occurs and the sleepiness may happen. The blood glucose level is not on normal flow and the body imbalance takes place.

 

  • Role of Antioxidant Defense System
  • Protection Mechanism of antioxidant
  • Role of ROS
  • Clinical studies of oxidative stress
  • Experimental studies of oxidative stress

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